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½ÄÇ° ¡í Áö¹æ ¡í Áö¹æÁ¾·ù ºÒÆ÷ÈÁö¹æ : ¿À¸Þ°¡ 6, ½Ä¿ëÀ¯ Áö¹æ»ê Á¾·ù - Linoleic Acid - oleic Acid Æ÷ÈÁö¹æ vs ºÒÆ÷ÈÁö¹æ - Æ÷ÈÁö¹æ À¯ÇØ·ÐÀÇ ¹®Á¦Á¡ : MCT ´Ù°¡ ºÒÆ÷È Áö¹æ - ºÒÆ÷È Áö¹æÀÇ µ¶¼º : ºü¸¥ »êÆÐ - ¿À¸Þ°¡ 6 À§Ç輺 - ¿À¸Þ°¡ 3 ±â´É: ¿À¸Þ°¡ 3 ¿¡ ´ëÇÑ Áö³ªÄ£ ±â´ë - CLA °ø¾×¸®³î·»»ê - ÇÁ·Î½ºÅ¸±×¶õµò -> ¾Æ¶óÅ°µ·»ê : Ç÷¾×ÀÀ°í Æ÷ÈÁö¹æ À¯Çطп¡ ´ëÇÑ ¹Ý·Ð ºÒÆ÷ÈÁö¹æ(¿À¸Þ°¡-6) °úÀ×¼·ÃëÀÇ À§Ç輺À» °æ°íÇÑ Ã¥ ¿À¸Þ°¡6 Áö¹æ»êÀÇ ¿ªÇÒÀº ¹«¾ùÀϱî¿ä? - Ç÷¾× ÄÝ·¹½ºÅ×·Ñ ¾çÀ» ³·ÃçÁÝ´Ï´Ù. - Ç÷°ü¼öÃà¿Í Ç÷¾×ÀÀ°í¸¦ µ½½À´Ï´Ù. - ¾Ë·¹¸£±â¿Í ¿°Áõ, Ç÷ÀüÀ» ÃËÁø½Ãŵ´Ï´Ù. - °úÀ×¼·Ãë´Â Ç÷¾ÐÀ» Áõ°¡½ÃÅ°°í ¸¸¼º¿°ÁõÀ» À¯¹ß½Ãŵ´Ï´Ù. ¸¸¼º ¿°ÁõÀÌ ¸¸º´ÀÇ ¿øÀÎÀ̶õ »ç½ÇÀ» µé¾îº¸¼Ì³ª¿ä? ¿À¸Þ°¡6 Áö¹æ»êÀ» °úÀ× ¼·ÃëÇÏ¿© ¹ß»ýµÇ¾îÁö´Â ºñ¸¸Àº ¸ö ÀüüÀÇ ¸¸¼º ¿°ÁõÀ» À¯¹ßÇÕ´Ï´Ù. ¸¸¼º ¿°ÁõÀº Áö¹æ¼¼Æ÷, ¸é¿ª¼¼Æ÷, ³ú¼¼Æ÷ µîÀ» ¸Á°¡¶ß·Á Àν¶¸° ÀúÇ×Áõ, ´ç´¢, µ¿¸Æ°æÈÁõ, ¾ËÃ÷ÇÏÀ̸Ó, ÆÄŲ½¼º´, ÇåÆÃÅϺ´, ¶Ç ¾ÏÀ» À¯¹ßÇÕ´Ï´Ù. ¿À¸Þ°¡6 Áö¹æ»êÀº µÎ°¡Áö·Î ºÐ·ùÇÒ ¼ö ÀÖ½À´Ï´Ù. - ¸®³î·¹»ê Linoleic acid - ¾Æ¶óÅ°µ·»ê Arachinonic acid ½Ä¹°¼º±â¸§ÀÎ ¿Á¼ö¼ö±â¸§, Çعٶó±â¾¾À¯, Äá±â¸§, Âü±â¸§¿¡¼ ¼·Ãë ÇÒ ¼ö ÀÖ½À´Ï´Ù. Pharmacology The conversion of cell membrane arachidonic acid (20:4n-6) to omega-6 prostaglandin and omega-6 leukotriene eicosanoids during the inflammatory cascade provides many targets for pharmaceutical drugs to impede the inflammatory process in atherosclerosis,[16] asthma, arthritis, vascular disease, thrombosis, immune-inflammatory processes, and tumor proliferation. Competitive interactions with the omega-3 fatty acids affect the relative storage, mobilization, conversion and action of the omega-3 and omega-6 eicosanoid precursors (see Essential fatty acid interactions). Suggested negative health effects Some medical research suggests that excessive levels of certain omega-6 fatty acids relative to certain omega-3 fatty acids may increase the probability of a number of diseases.[17][18][19] Modern Western diets typically have ratios of omega-6 to omega-3 in excess of 10 to 1, some as high as 30 to 1; the average ratio of omega-6 to omega-3 in the Western diet is 15:1–16.7:1.[16] Humans are thought to have evolved with a diet of a 1-to-1 ratio of omega-6 to omega-3 and the optimal ratio is thought to be 4 to 1 or lower,[16][20] although some sources suggest ratios as low as 1:1.[21] A ratio of 2–3:1 omega 6 to omega 3 helped reduce inflammation in patients with rheumatoid arthritis.[16] A ratio of 5:1 had a beneficial effect on patients with asthma but a 10:1 ratio had a negative effect.[16] A ratio of 2.5:1 reduced rectal cell proliferation in patients with colorectal cancer, whereas a ratio of 4:1 had no effect.[16] Excess omega-6 fatty acids from vegetable oils interfere with the health benefits of omega-3 fats, in part because they compete for the same rate-limiting enzymes. A high proportion of omega-6 to omega-3 fat in the diet shifts the physiological state in the tissues toward the pathogenesis of many diseases: prothrombotic, proinflammatory and proconstrictive.[22] Chronic excessive production of omega-6 eicosanoids is correlated with arthritis, inflammation, and cancer. Many of the medications used to treat and manage these conditions work by blocking the effects of the COX-2 enzyme.[23] Many steps in formation and action of omega-6 prostaglandins from omega-6 arachidonic acid proceed more vigorously than the corresponding competitive steps in formation and action of omega-3 hormones from omega-3 eicosapentaenoic acid.[24] The COX-1 and COX-2 inhibitor medications, used to treat inflammation and pain, work by preventing the COX enzymes from turning arachidonic acid into inflammatory compounds.[25] (See Cyclooxygenase for more information.) The LOX inhibitor medications often used to treat asthma work by preventing the LOX enzyme from converting arachidonic acid into the leukotrienes.[26][27] Many of the anti-mania medications used to treat bipolar disorder work by targeting the arachidonic acid cascade in the brain.[28] A high consumption of oxidized polyunsaturated fatty acids (PUFAs), which are found in most types of vegetable oil, may increase the likelihood that postmenopausal women will develop breast cancer.[29] Similar effect was observed on prostate cancer, but the study was performed on mice.[30] Another "analysis suggested an inverse association between total polyunsaturated fatty acids and breast cancer risk, but individual polyunsaturated fatty acids behaved differently [from each other]. [...] a 20:2 derivative of linoleic acid [...] was inversely associated with the risk of breast cancer".[31] |
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